Yeah, the GeparOLA study we present here is the long term data and we have already published the pathological complete response data in 2020 in Annals of Oncology, which demonstrated that exchanging carboplatin with olaparib in patients who have HRD, so defined as either BRCA mutation or having a HRD-high tumor by the MyChoice test demonstrate that the pCR rate is not different between the two agents...
Yeah, the GeparOLA study we present here is the long term data and we have already published the pathological complete response data in 2020 in Annals of Oncology, which demonstrated that exchanging carboplatin with olaparib in patients who have HRD, so defined as either BRCA mutation or having a HRD-high tumor by the MyChoice test demonstrate that the pCR rate is not different between the two agents. So basically, olaparib can be used instead of carboplatinum, looking at the pCR rate. But we also want to know how are the long-term outcome data? And here we present the long-term outcome data from the GeparOLA study and the overall cohort where we have, of course, mutant versus wild-type patients demonstrated that in the long run, carboplatinum is better than olaparib. But when we look at the BRCA carriers, there is no difference. The only difference we see is in the wild-type cohort, which still have HRD-high tumors, but they don’t have a mutation in the tumor and here we see the difference between carboplatinum with olaparib.